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heat-stress-response

1 article

Heat-stress-response encompasses the physiological and molecular mechanisms plants employ to tolerate and survive exposure to elevated temperatures. This research is critical as climate change intensifies heat stress globally, threatening agricultural productivity and natural ecosystems. Understanding how plants perceive, signal, and respond to heat stress is essential for developing crops resilient to future climate conditions.

TuATG1-mediated autophagy confers thermotolerance in Tetranychus urticae and provides an RNAi target for pest management.

PubMed · 2026-02-19

Researchers discovered that a heat-tolerant agricultural pest, the spider mite Tetranychus urticae, depends on an autophagy gene (TuATG1) to survive hot conditions. By silencing this gene with RNAi technology, they achieved 100% mortality at high temperatures, offering a promising new pest control method that could reduce reliance on chemical pesticides.

1

Heat stress at 39°C triggers broad transcriptional reprogramming, with lysosomal and autophagy pathways most significantly enriched

2

TuATG1 silencing achieved 76.36% knockdown and caused 31.47% mortality at 25°C, with complete mortality after 5 hours at 42°C

3

TuATG1 is critical for both basal homeostasis and heat adaptation, making it a viable RNAi target for sustainable pest management